I heard about Ginkgo Biloba being known for acting like a "natural blood thinner". Perhaps that's my go-to?
The doctors I go to refuse to prescribe me Pentox. Other than that, I'm only using NSAIDS and nightly Cialis to treat my Peyronie's.
Is there a worry of nightly Cialis causing priapism? I use 2,5-5mg/night. Perhaps I should start taking the Cialis in the morning instead. Does anyone here do that?
I wish some of you guys on here could shed some lights on my questions and concerns.
Any ideas guys? It would be much appreciated! Thanks!
Regarding Ginkgo Biloba, you can have a look at Table 2 in following study - it compares molecular mechanism of various substances including Pentox and Ginko Biloba. I have also read somewhere on the forum, that these two have indeed some similar effects (cannot find this post now). Hope this helps.
https://www.mdpi.com/1422-0067/23/24/15969
Thank you very much. I'm not finding the section regarding Ginkgo Biloba though. Could you copy & paste the part where it is mentioned?
You need to click on Table 2 to display.
| Ginko Biloba | - Antioxidant
- Anti-inflammatory
- Antifibrotic
- Vasorelaxant
- Antiplatelet aggregation
| - It scavenges reactive oxygen species and reactive nitroxidative species.
- It reduces production of: i-NOS, NF-kappa-B, IL-1, IL-6, IL-10, TNF-α, TGF-β1, and PAI-1
- It hinders inflammation, inhibiting: NF-kappa-B and pro-inflammatory cytokine production, and COX-2 activity
- It inhibits fibrosis (deposition of collagen) by inhibiting TGF-β1
- It determines vasodilation through an endothelial mechanism, via the nitric oxide pathway.
- It inhibits platelet aggregation
|
|
| Pentoxyfiline | - Antioxidant
- Anti-inflammatory
- Antifibrotic
- Vasorelaxant
- Antiplatelet aggregation
| - It scavenges reactive oxygen species and reactive nitroxidative species.
- It reduces production of: TNF-α, i-NOS, IL-1, IL-6, IL-8, IL-10, TGF-ß1, PDGF, and PAI-1.
- It hinders inflammation, inhibiting: NF-kappa-B and pro-inflammatory cytokine production.
- It inhibits fibrosis (deposition of collagen) by inhibiting TGF-β1 and PDGF.
- It determines vasodilation through a nonselective PDE inhibition (preventing the degradation reaction of cyclic AMP).
- It inhibits platelet aggregation.
|