Which is the best natural substitute for Pentox?

[desmondthethird]

I heard about Ginkgo Biloba being known for acting like a "natural blood thinner". Perhaps that's my go-to?
The doctors I go to refuse to prescribe me Pentox. Other than that, I'm only using NSAIDS and nightly Cialis to treat my Peyronie's.
Is there a worry of nightly Cialis causing priapism? I use 2,5-5mg/night. Perhaps I should start taking the Cialis in the morning instead. Does anyone here do that?

I wish some of you guys on here could shed some lights on my questions and concerns.

Any ideas guys? It would be much appreciated! Thanks!  

[martin111]

Regarding Ginkgo Biloba, you can have a look at Table 2 in following study - it compares molecular mechanism of various substances including Pentox and Ginko Biloba. I have also read somewhere on the forum, that these two have indeed some similar effects (cannot find this post now). Hope this helps.

https://www.mdpi.com/1422-0067/23/24/15969


 

[desmondthethird]

Thank you very much. I'm not finding the section regarding Ginkgo Biloba though. Could you copy & paste the part where it is mentioned?
 

[martin111]

You need to click on Table 2 to display.

Ginko Biloba	Antioxidant Anti-inflammatory Antifibrotic Vasorelaxant Antiplatelet aggregation	It scavenges reactive oxygen species and reactive nitroxidative species. It reduces production of: i-NOS, NF-kappa-B, IL-1, IL-6, IL-10, TNF-α, TGF-β1, and PAI-1 It hinders inflammation, inhibiting: NF-kappa-B and pro-inflammatory cytokine production, and COX-2 activity It inhibits fibrosis (deposition of collagen) by inhibiting TGF-β1 It determines vasodilation through an endothelial mechanism, via the nitric oxide pathway. It inhibits platelet aggregation
Pentoxyfiline	Antioxidant Anti-inflammatory Antifibrotic Vasorelaxant Antiplatelet aggregation	It scavenges reactive oxygen species and reactive nitroxidative species. It reduces production of: TNF-α, i-NOS, IL-1, IL-6, IL-8, IL-10, TGF-ß1, PDGF, and PAI-1. It hinders inflammation, inhibiting: NF-kappa-B and pro-inflammatory cytokine production. It inhibits fibrosis (deposition of collagen) by inhibiting TGF-β1 and PDGF. It determines vasodilation through a nonselective PDE inhibition (preventing the degradation reaction of cyclic AMP). It inhibits platelet aggregation.