Do you still get a spontaneous tingle when you see a beautiful Woman?

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Andy_75

Hawk,

I can't wrap my head around the disconnect between the psychological and physiological gap with an implant?

What I mean is, an average man can get an erection in 2 ways and in most men, it is a combination of the two.  
1.  Being mentally/psychologically turned on and
2.  A bodily reflex.

So, you have an implant, you are with a female, Your libido is high...you are turned on mentally, both visually and physically through touch and seeing the woman..,does anything happen down there?  Like those tingles in the glans/head, precum?

Because from what I have read, once you pump up, the skin, glans/head feel the same as always but what about prior to or during foreplay?

I am just trying to wrap my head around the inability to have a natural erection because it is very much a mind & body connection or experience.  

Does that mind & body connection/experience remain intact once you pump up?  Do you feel at all "artificial" at times or like your penis is missing?  

I mean, just now, I close my eyes, picture a woman naked, my penis tingles, moves a little, slightly engorges and this in turn feeds my mental fantasy and sex drive, the tingles and feeling of my penis beginning to engorge between my legs and the mental/physical begin to snowball.

What about orgasm?  Does it feel the same or is it different?  

I have been reading and searching but it would be great if you could address these if possible as your answers are always so informative and descriptive.

Thanks a lot!

Age: 40
- Symptoms: 2 Bumps on shaft (1 top/1 bottom) Jan 2021
- No pain/minor dorsal curve/subtle dent across width of shaft creates pliable erection with weak hinge like effect/mid shaft).

Hawk

Andy, I will try my best to answer you in detail but before I answer your questions I need a couple of answers from you.

1. Do you have ED? If so to what degree

2. Do you get "tingles" down there independent of your penis starting to get erect or is it associated with the process of starting to get erect?

2. Can you reliably have intercourse that is satisfying to you and your partner?

Prostatectomy 2004, radiation 2009, currently 70 yrs old
After pills, injections, VED - Dr Eid, Titan 22cm implant 8/7/18
Hawk - Updated 10/27/18 - Peyronies Society Forums

Andy_75

Hey Hawk,

1. No, I can still get it up.

2. Both, I get tingles if I see an attractive woman or engage in foreplay and during the process of starting to get an erection.

3. I have not tried but I do not believe so.  Unfortunately, I have developed a slightly indented line across my penis due to the small lump in the middle of my penis on the top/dorsal (I am assuming). This means, that although I can get an erection, I am extremely nervous to even attempt sexual intercourse or masturbation due to the risk of "buckling" along that slightly indented line across my penis which has made my erection pliable and weak/bendable at that point.  I hope that makes sense.  Think of a Cucumber if you lacerated it with a knife about 1/4 deep across the body –so there is a weak point that could cause the cucumber to snap under pressure.

I am trying to ride this acute phase out (take the supplements I can, exercise, good diet etc.) and hope things improve but am also curious and exploring options if things were to worsen or not improve.

Thanks and I look forward to your response.
Age: 40
- Symptoms: 2 Bumps on shaft (1 top/1 bottom) Jan 2021
- No pain/minor dorsal curve/subtle dent across width of shaft creates pliable erection with weak hinge like effect/mid shaft).

Stepone

Andy, ouch!
I had the same problem, except mine was on the left side.
My urologist called it a hinge. It healed by just as the name indicated, it acted as a hinge allowing my penis to bend.
I had surgery to fix it, but it shortened my 8+ inch penis to 6.5". And my girth shrank too. A few years later, I received an implant. I wished I had the implant first and bypassed the corrective surgery.
Good luck in your quest, but If I were you, I would find a urologist that specializes in penis issues. Don't go to a urologist that is a general urologist. I had to visit 5 urologists to find a specialist.
Check out the doctors on this website.
Explore all your options, you are still young and have a lot of years ahead of you.
StepOne  
Nesbit surgery 2015, 66 years young, Titan Implant 4/25/19, 22cm, Dr. Lentz, Duke University NC

Hawk

Well, Andy, you asked for it!  Here it is!

I don't say this to boast but to give a clear answer.  I do not think any man was ever more spontaneous or enduring than I was for most of my life.  Even after decades of marriage, if I walked in the bedroom and my wife was in bed, all she had to say was some form of "do you want to play."  Before I could kick my shoes off and drop my pants, I had a diamond-cutter of an erection.  I would typically have two orgasms with the same erection, and once or twice a year, I would have up to 5 orgasms without ever losing an erection.  Never in my life did I lose an erection while my penis was inserted, even if I could not have another orgasm.  That was true even back when I used to drink alcohol, although I never got drunk enough to pass out. I have gone through periods where I have had sex every day for at least two months straight.

Since it appears that this is what you asked for, that was my typically long, detailed way to put the next statement in perspective. I have never experienced "a tingling feeling or any other feeling in my penis from looking at or thinking of a woman unless I got an erection.  I certainly could not do much more than a long glance without getting an erection, however. So the concept of a tingling in my penis that is not associated with my penis starting to fill is totally foreign to me.

Now, let's use some reasoning. Any sensation in a penis, whether good or bad, has to be the result of nerves.  Blood certainly has no feeling.  If your nerves are exactly like they were before an implant, how could an implant possibly make your penis feel differently?  If you get a "tingle in your penis" the instant you look at a woman, how could an implant in your penis block that?  If you don't lose ANY tissue during an implant, then all the nerves are available to feel whatever the brain and nerves felt before.  In fact, if you opted to get an implant from a great surgeon, then you should not only expect to feel the same, you should expect that when you think of a woman that your glans and spongiosum will fill with blood to the degree they did just before surgery.

As great as an implant is, it is not rocket science, at least not in theory.  Your cavernousa "chambers" are engorged with warm saline rather than warm blood.  A lining is placed in the chamber so the saline is not lost and can be returned to the reservoir for the next time you want it.  Common-looking tubes and a tiny pump enable you to move the saline to either the penis or the reservoir at will.

Your erect penis still feels heavy and full because it is.  You will, however, never have a fully erect penis without 3-25 quick pumps,  no matter what you look at.  The difference is in the process, not in the feeling.

All this, of course, assumes some hack does not cut a nerve while doing a retro-pubic approach, but I think that is a risk, in theory, more than in actuality.

PS: Frankly, I have to think your statement about tingling in your penis without any beginning of an erection is dubious.  I think you get a psychological tingling in your brain. Your heart "skips a beat," etc.  I respectfully doubt that your penis tingles. What could that be?  Your nervous system does not work like that.  Your nerves sense touch and change (engorgement).  They do not create sensation.  They transmit sensation.



Prostatectomy 2004, radiation 2009, currently 70 yrs old
After pills, injections, VED - Dr Eid, Titan 22cm implant 8/7/18
Hawk - Updated 10/27/18 - Peyronies Society Forums

Mikel7

Lump 4/2020, age 62 , Dr Levine 6-26-20, Dors Curve 11/2020, Peyronies
Vit E400mg, COQ10, Heat Therapy, Penimaster, Pentox, Cialis, Restorex
SNHL 7/2020 - Stopped all Meds because ototoxicity  Heat/traction/VED are working. CPPS Diagnosis - Stable :)

Andy_75

Thanks a lot Hawk!

Maybe tingly isn't the right word.  I can get like a feeling of different but good sensations in different parts of the glans and shaft, the tip being most sensitive, prior to an erection when I am turned on.

Maybe it is just me.  Heh.

Thanks for the advice Stepone.
Age: 40
- Symptoms: 2 Bumps on shaft (1 top/1 bottom) Jan 2021
- No pain/minor dorsal curve/subtle dent across width of shaft creates pliable erection with weak hinge like effect/mid shaft).

Hawk

I have trouble accepting that without any slight touch and without starting to fill with blood, your brain starts sending sensations to your penis.

BUT, if it does, there is certainly nothing about an implant that would stop that.
Prostatectomy 2004, radiation 2009, currently 70 yrs old
After pills, injections, VED - Dr Eid, Titan 22cm implant 8/7/18
Hawk - Updated 10/27/18 - Peyronies Society Forums

Andy_75

You and others may find this article below interesting.  It talks about the connection between the brain, CNS (Central Nervous System), Autonomic Nervous System, Parasympathetic Neurons and how an erection is a carefully orchestrated series of events controlled by these systems.

As their Working Group colleague William D. Steers has noted, any disturbance in the network of nerve pathways that connects the penis and the CNS can lead to problems with erections.

So I guess my questions were more along the lines of what is it like to see a female or have a female touch you (fantasizing, kissing, light foreplay etc.) and feeling nothing in your penis until you turn on the device and she physically touches your penis?  Or do you feel anything in your penis at all when mentally fantasizing or when touching/kissing a female/partner?  

The implant surgery disturbs the network of nerve pathways that connects the penis and the CNS.  With many surgeons stating that, "the implant surgery makes it impossible to ever have a "latent" or natural erection that's not dependent on the device."

I suppose that I am just curious to how that feels?  It being impossible to ever have a "latent" or natural erection that's not dependent on the device, both physically and psychologically/mentally?  Is it difficult to adapt to?  Not too big of a deal?  Obviously, if you have complete ED from severe Peyronie's or a spinal cord issue etc, it's a non issue because the feeling down there is pretty much gone with the disconnect between your brain and penis already being there but what if you still have partial feeling?  If you can still get erections but they are not strong enough or last long enough for intercourse?

If I am not explaining myself correctly or clearly, I apologize and don't worry about answering the questions.

Here is the article in case you or any others are interested in reading it.

QuoteThe Central Mechanisms of Sexual Function
by Irwin Goldstein, MD


The management of erectile dysfunction (ED) has changed dramatically in recent years, as advances in molecular biology have given us a better understanding of the erectile process as well as the pathophysiology of erectile disorders. Until relatively recently, however, most research in ED focused on peripheral neurophysiology and on the local tissues of the penis, leading to the development of highly effective treatments such as penile injections and sildenafil. There has, however, been growing interest in the role of the central nervous system (CNS) in the control of erectile function, and researchers have begun to develop medications that target these central mechanisms. The first of these agents is apomorphine, a drug that has been used for over a century for the treatment of Parkinson's disease and other disorders. Researchers began evaluating apomorphine as a potential treatment for ED in the mid 1980s, and it is currently under review by the Food and Drug Administration. Unlike sildenafil, which acts only on tissues in the penis, apomorphine acts directly on the brain.

In response to the development and impending availability of apomorphine as a centrally acting agent for ED, a consensus group known as the Working Group for the Study of Central Mechanisms in Erectile Dysfunction was formed in 1998 to examine how the brain and spinal cord control penile erections. Members of this group include Irwin Goldstein, Boston University, John Bancroft of Indiana University, François Giuliano of the Faculté de Médecine, Université Paris-Sud, Jeremy P. W. Heaton of Queen's Universiry, Ontario, Ronald W. Lewis of the Medical College of Georgia, Tom F. Lue of the University of California, San Francisco, Kevin E. McKenna of Northwestern University, Harin Padma-Nathan of the University of Southern California, San Francisco, Raymond Rosen of the Robert Wood Johnson Medical School, Benjamin D. Sachs of the University of Connecticut, R. Taylor Segraves of Case Western Reserve University, and William D. Steers of the University of Virginia. The group met recently to discuss and review what is presently known about these mechanisms and to consider future areas of research. The following are some of our findings and conclusions.

An erection is a carefully orchestrated series of events controlled by the CNS. We now know that the penis is under the complete control of the CNS, both during sexual arousal and at rest. As our Working Group colleague William D. Steers has noted, any disturbance in the network of nerve pathways that connects the penis and the CNS can lead to problems with erections.

The male sexual response reflects a dynamic balance between exciting and inhibiting forces of the autonomic nervous system within the penis and throughout the CNS. The sympathetic component tends to inhibit erections, whereas the parasympathetic system is one of several excitatory pathways. During arousal, excitatory signals can originate in the brain, either by the sight or thought of an appealing sexual partner or by physical genital stimulation. Regardless of the source of these signals, the excitatory nerves in the penis respond by releasing proerectile neurotransmitters such as nitric oxide and acetylcholine. These chemical messengers signal the smooth muscles of the penile arteries to relax and fill with blood, resulting in an erection. The drug sildenafil works directly on the tissue in the penis to keep muscles relaxed and the vessels engorged.

Many regions in the brain contribute to male sexual response, ranging from centers in the hindbrain that also regulate basic body functions such as breathing, to areas in the cerebral cortex, the organ that controls higher thought and intellect. Research demonstrates that no single area of the brain controls sexual function. Rather, control is distributed throughout multiple areas of the brain and spinal cord. Should injury or disease destroy one or more of these regions, the ability to have erections often remains intact.

Switching off the activity of the sympathetic nervous system enhances erections. Nocturnal erections are a good example of this. Nocturnal erections occur primarily during rapid eye movement (REM) sleep, the stage in which dreaming occurs. During REM sleep, sympathetic neurons are turned off in the locus coeruleus, a specific area of the brain stem. According to one theory, when the sympathetic nervous system is at rest, proerectile pathways predominate and allow nocturnal erections to occur. We often refer to these events as a "battery-recharging" mechanism for the penis, because they increase blood flow to the penis and thus bring oxygen to reenergize it. Studies show that women also experience episodes of nocturnal arousal when the sympathetic nervous system is a rest. Approximately four or five times a night, or during each period of REM, women experience labial, vaginal, and clitoral engorgement.

Some erections may be generated entirely by the spinal cord. Evidence for these "reflexive" –type of erections comes from observations on World War II soldiers with spinal cord injuries. Prior to these observations, it was generally believed that men with spinal cord injuries had permanent and complete ED. We now know that this view is mistaken. Studies in men with severe or complete spinal cord injury have demonstrated that many men were able to achieve erections and engage in vaginal penetration even though their injuries left them unable to control other bodily functions. These observations, as well as information from studies in laboratory animals as far back as the 1890s, led to the discovery of an erection-generating center located in the sacral segments of the spinal cord (between the S3 and T12 vertebrae). Researchers found that physical stimulation of the penis sends sensory signals via the pudendal nerve to this erection center. Incoming signals activate connector nerve cells (interneurons) to stimulate nearby parasympathetic neurons. These neurons then transmit erection-inducing signals from the sacral spine to the penile blood vessels. As long as this reflex arc remains intact, an erection is possible.

Observations of men and laboratory animals with spinal cord damage indicate that when the brain is disconnected from the erection-generating center in the spinal cord, erections generally occur more often and with less tactile stimulation than before the injury. Studies in rats by Group member Benjamin D. Sachs led to the theory that disconnecting the brain from the body, removed some inhibitory control over erections. This proved to be the case, as demonstrated by physiologist Kevin E. McKenna, also a member of our Working Group. In 1990, McKenna and his colleague Lesley Marson identified the area of the brain that controls spinal-mediated erections. This cluster of neurons in the hindbrain (an evolutionary ancient part of the brain that controls blood pressure and heart rate) is called the paragigantocellular nucleus (PGN). The investigators found that the PGN neurons send most of their axons down to the erection-generating neurons in the lower spinal cord. There the PGN neurons release the neurotransmitter serotonin, which inhibits erections by opposing the effects of proerectile neurotransmitters.

This discovery may have important implications for people who take drugs that enhance levels of serotonin, such as the selective serotonin reuptake inhibitors (SSRIs) that are used to treat depression and other mental health disorders. These drugs often cause sexual dysfunction as a side effect, most commonly delayed or blocked ejaculation in men and a reduced sexual desire and difficulty reaching orgasm in women. The work by McKenna and Marson helps explain how this common and troublesome SSRI side effect may occur. By increasing levels of serotonin in the CNS, the SSRIs may tighten the brain's built-in controls on erection, ejaculation, and other sexual functions. Interestingly, however, some recent studies also suggest that the inhibiting effects of the SSRIs may actually be helpful for some patients with other types of sexual dysfunction, such as premature ejaculation or inappropriate or excessive sexual urges.

Inhibitory control over sexual behavior may be a protective mechanism for humans. Considering the importance of sex to the preservation of the species, it is not clear why these elaborate inhibitory controls have evolved. One theory by Group member John Bancroft suggests that for most men this central inhibition is adaptive and helps keep them out of trouble resulting from excessive sexual activity or high-risk sexual behavior. These internal controls may also prevent men from experiencing repeated ejaculations during sexual encounters, which could lower sperm counts and thus reduce fertility. Despite these potential benefits, Bancroft believes that too much central inhibition, such as from high levels of serotonin, could result in unwanted sexual dysfunction.

The hypothalamus plays an important role in regulating sexual behavior. This region of the brain links the nervous and endocrine systems and is involved in certain basic behaviors such as eating and aggression. A cluster of neurons in the hypothalamus called the medial preoptic area (MPOA) appears to play a critical role in sexual function and is being intensively studied at the moment. Group member François Guiliano and his colleagues have recently shown that electrical or chemical stimulation of the MPOA causes erections in rats. The MPOA appears to integrate stimuli from many areas of the brain, helping to organize and direct the complex patterns of sexual behavior. The hypothalamus also contains the paraventricular nucleus. Like the MPOA, the paraventricular nucleus acts as a processing center that sends and receives messages from different parts of the brain and spinal cord. The erection-enhancing effects of apomorphine occur when it mimics the actions of the neurotransmitter dopamine and binds to specific receptors in the paraventricular nucleus and the MPOA, thereby turning on proerectile pathways.

During sexual arousal, the paraventricular nucleus also releases oxytocin, the hormone that stimulates uterine contractions during labor, as well as the release of milk during breast-feeding. We now know that oxytocin is also an important neurotransmitter in men, with powerful proerectile effects, as it activates excitatory nerve pathways from the spinal erection-generating center to the penis.

In addition to the above discoveries and advances, we are also exploring how higher brain functions such as memory and learning help to control erections. Group member Raymond Rosen has recently shown that healthy men can be taught to have erections on demand in response to mental imagery or nonsexual cues.

We have also become aware of the many similarities and differences between the sexes regarding CNS control of arousal, orgasm, and various sexual functions, although this area of research in women lags far behind that in men. We hope that a further understanding of the role of the brain and spinal cord in controlling sexuality will lead to the development of more effective treatments for both male and female sexual dysfunction.
Age: 40
- Symptoms: 2 Bumps on shaft (1 top/1 bottom) Jan 2021
- No pain/minor dorsal curve/subtle dent across width of shaft creates pliable erection with weak hinge like effect/mid shaft).

Stepone

Ok I can only speak for me.
I received an implant because I could not maintain an erection. With my implant, I gained girth and length, and lost my hourglassing. I did not go to Dr Eid or Dr Kramer, but I did go to a high volume implant surgeon.
If I see some hot pics or start kissing or think about sex, I still feel a tingle in my penis and yes I will get a semi erect shaft and penis head.
The head of my penis will get fully engorged. It doesn't stay that way without constant stimulation, and as I said, I still have leakage.
But yes I still feel the excitement in my penis.
My penis is not dead, it is just super charged now, lol
StepOne  
Nesbit surgery 2015, 66 years young, Titan Implant 4/25/19, 22cm, Dr. Lentz, Duke University NC

Hawk

Andy, I almost sympathize with you because no answer is good enough or satisfying to you.  It only raises more questions and sends you searching for even more reinforcing evidence.  I sympathize because It reminds me so much of myself.  I can make charts and graphs, Pro-Vs-Con lists, and ask questions until I annoy multiple sources.

First is this.  NONE of this matters because people get an implant because they cannot have any confidence in being able to have intercourse.  You are straining at gnats.  Stop for a moment and compare these: 1. The nuance of being able to feel a little tingle in your penis when a woman looks in your eyes in a way that says, "take me! I'm yours!"  you feel a rush of panic and blurt out, "sorry, I have to go.  I can not have intercourse." then rush out the door. VS. 2.  You take her home, make mad passionate love to her, including rough vigorous sex with an explosive mind-blowing orgasm.  Then, because of a deep connection you feel, you make long slow tender love to her for hours.  She spends the night and is so enamored by the experience she reaches for you as she wakes with the first light of morning.  You willingly and confidently quench the desire you have sparked with your previous performance.  You have another memorable intense climax with her gasping in your arms.

Yes, I know I should write cheap steamy romance novels.  ;D  

BUT that is the comparison.  So, even IF guys without an implant get a tingle in their penis before they run out the door that men with implants don't get until they pump up and have a sex-fest, WHO CARES?  How would that be useful information or have ANY bearing on the decision-making process?  It wouldn't.  It is a useless exercise in the absurd.  

How can I make any more all-inclusive definitive statement than to say; My penis feels just like it did when I was 20 (both flaccid and erect) EXCEPT, I never have an embarrassing unwanted erection.

I went through a period of total Erectile Dysfunction unless I had drugs to induce one.  I cannot tell you how many times I was aroused so much and felt JUST LIKE I HAD FOR DECADES BEFORE.  It was so strong, so familiar I was certain I had to have an erection only to reach down and none.  Men so misunderstand their sexual organs and the association or their penis with other sexual events.  I have had some of the most intense orgasms of my life, TOTALLY FLACCID.  The aura you attach to a spontaneous erection is misplaced.

After this discussion, maybe we can discuss how love really feels so those who have never experienced love with know for sure when they do.  Or maybe we could have a woman explain to us EXACTLY how intercourse feels so we will perfectly know the feeling to her as well as she knows the feeling herself.  The point is there are limits to the degree to which you can explain an experience to someone who is limited to imagining that experience.  You have reached that limit.  

Prostatectomy 2004, radiation 2009, currently 70 yrs old
After pills, injections, VED - Dr Eid, Titan 22cm implant 8/7/18
Hawk - Updated 10/27/18 - Peyronies Society Forums

Andy_75

Stepone,

Thanks a lot for your comment.  Great answer and that is exactly what I was wondering and curious about.

Cheers,
Age: 40
- Symptoms: 2 Bumps on shaft (1 top/1 bottom) Jan 2021
- No pain/minor dorsal curve/subtle dent across width of shaft creates pliable erection with weak hinge like effect/mid shaft).

Andy_75

Thanks again Hawk for taking the time to respond.  I understand what you are getting at, it's about the big picture and not so much the little nuances.

Sorry if I have frustrated you.  It was never my intention.  I am just curious about certain things as I explore the penile implant option.

I understand your well made point.  For that, I am appreciative.

Cheers,
Age: 40
- Symptoms: 2 Bumps on shaft (1 top/1 bottom) Jan 2021
- No pain/minor dorsal curve/subtle dent across width of shaft creates pliable erection with weak hinge like effect/mid shaft).

Hawk

Andy, I admit to being a little frustrated but affectionately so.  I also admit to filling my wife in on your quest for answers and I read my response to her.  She has lived with some of that so she could appreciate it. :)

I had several typos in my post so you might want to re-read it.

Cheers
Prostatectomy 2004, radiation 2009, currently 70 yrs old
After pills, injections, VED - Dr Eid, Titan 22cm implant 8/7/18
Hawk - Updated 10/27/18 - Peyronies Society Forums

TDix

Like Step, I too can get semi erect without a single pump if aroused.  The shaft and glans still fill with blood, but it is not hard enough for sex.  Just a few pumps however, and I'm good to go.  An implanted penis is not "dead".  It is still very much connected to your body and feels no different than it did for me 20 years ago, other than a pump in the scrotum.  Yes, I know there is an implant in there, but even my wife of over 20 years would not know the difference if she never knew, again other than the pump.....and even then it takes a firm grasp on the scrotum to feel it
47 yrs old, 3 yrs diagnosed
Xiaflex w/original uro resulted in a fracture
Excision/grafting by Dr Faysal Yafi 3/26/19
Implanted by Dr Yafi 8/11/20, Titan 20cm + 1cm RTE

Kobegianna

These posts are so convincing toward getting an implants. Yet it is so hard to take the plunge. I honestly think it may possibly be better to have complete dysfunction so only option is an implant rather than a half functioning penis which makes taking the plunge to get an implant much harder.  
34 yrs Old, Healthy very active
Possibly injured penis or took too much Cialis
Symptoms starting January-February 2021
First dent seen April 4, 2021, painful erections

TDix

For me, and this was solely my decision, I wanted the nightmare over-whatever it took.  That ended up being an implant after trying other options.  Point is, do you want to live with it or do you want to do all you can to fix it and move on.  If you have a knee injury do you live with a limp and pain the rest of your life or do you get it fixed?  Yes an artificial knee is definitely different than what you were born with, and may ache in cold weather, may limit certain motions, etc... but it fixes the problem.  That really is a summary of what an implant does, it fixes a problem, as simple as that.  Yes, for younger men the thought of revisions down the line are a consideration, but the thought that for a good 10-15 years you don't have the worry and depression of having a part of your life taken from you is almost priceless.  I will more than likely need a revision, but that is a drop in the bucket, in my view, to years of a functional penis
47 yrs old, 3 yrs diagnosed
Xiaflex w/original uro resulted in a fracture
Excision/grafting by Dr Faysal Yafi 3/26/19
Implanted by Dr Yafi 8/11/20, Titan 20cm + 1cm RTE

Andy_75

Great comments and thanks for the specific info regarding your personal experience with an implant TDix.

Kobegianna,

I can completely relate to what you are saying, 100%.  Hence my inquiry/curiosity as to what men still do and do not feel once they get an implant.

You can read and read and understand the mechanism but you can't put a price on conversing with those who have had the procedure.  It really is great to be able to read experiences and I truly appreciate those who share what they still can and cannot feel.  For the most part, it would seem that there still is very much, a mind-body connection, especially if some can get semi erect which I had no idea was possible.  I am sure it is likely slightly different for everyone depending on surgeon and body chemistry etc.
Age: 40
- Symptoms: 2 Bumps on shaft (1 top/1 bottom) Jan 2021
- No pain/minor dorsal curve/subtle dent across width of shaft creates pliable erection with weak hinge like effect/mid shaft).

Hawk

Quote from: Andy_75 on July 06, 2021, 03:48:41 PM
...Hence my inquiry/curiosity as to what men still do and do not feel once they get an implant. There is nothing they do not feel.  feeling IS THE SAME

...some can get semi erect which I had no idea was possible.  I am sure it is likely slightly different for everyone depending on surgeon and body chemistry etc.  It has nothing to do with body chemistry or the surgeon

For clarity, a great high volume surgeon is important but whether you get semi-erections has very little to do with the surgeon or body chemistry.  It has to do with whether you could get erections before the surgery.  Your glans and spongiosium (the smaller center chamber around the urethra) will respond the same after an implant as they did before an implant.

Some men get implants only for straightening, and they have good erections, or they get partial erections before implant, or they get erections that fade if stimulation stops.  Their glans and spongiosium will respond the same after an implant as it did before an implant.

Also, remember.  This is really not a discussion about what you can and cannot feel.  I think every man healed from a properly installed implant will tell that they feel what they felt before.

I will one last time restate the facts.  Nothing is altered in the CNS during an implant.  From the penis sending signals to the brain and the brain and spinal cord sending signals back to the penis.  It is like asking how does a penis with a tattoo feel?? Or how does a circumcised penis feel when you see a woman.  It feels the same.  Nothing associated with the feeling process is altered.  Again, the difference is ONLY in the process of erection.  It has nothing to do with nerve pathways or feeling.
Prostatectomy 2004, radiation 2009, currently 70 yrs old
After pills, injections, VED - Dr Eid, Titan 22cm implant 8/7/18
Hawk - Updated 10/27/18 - Peyronies Society Forums

Andy_75

Hawk,

Great!  Thanks again for clarifying and reiterating things.  I fully understand now.  

Cheers,
Age: 40
- Symptoms: 2 Bumps on shaft (1 top/1 bottom) Jan 2021
- No pain/minor dorsal curve/subtle dent across width of shaft creates pliable erection with weak hinge like effect/mid shaft).

SW01

Yes. I do not even have to pump up unless I want penetrative sex. Oral. I do not need to pump up. Plenty hard and aroused.
Dealt with ED, Peyronies, & venous leak for 3 yrs.
implant on March 7, 2019 w/ a Titan 18 cm plus 1cm RTE
Revision after hernia surgery. Dr. Andrew Todd, Richmond KY
Removed Titan and put in LGX 18 cm plus 2 RTE's, 20 cm total.